Vascular Disease, Parkinson's, and Now Alzheimer's—Is Homocysteine the New All-Around Bad Guy?
levels and neuropathologically pure AD (Clarke et al., 1998). That is, the homocysteine-AD link seems to
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Untapped Resource? New Study to Boost Acceptance of CSF Analysis
mild cognitive impairment to AD (see Hansson et al., 2006; Fagan et al., 2006; Shaw et al., 2009; and
Leaky Blood-Brain Barrier a Harbinger of Alzheimer's?
Cullen et al., 2005; Goos et al., 2009; and Feb 2014 news). Iron, possibly from the breakdown of
How Cognitive Stimulation Might Protect Against Aβ Oligomers
accumulate less amyloid in the brain (see ARF related news story; Arendash et al., 2004; Jankowsky et al.,
Webinar: Axonopathy in Mouse Models of AD
transgenic mouse models (Casas et al., 2004). Some neurodegenerative diseases including AD and amyotrophic
Computational Modeling—Will it Rescue AD Clinical Trials?
simulation tool (CTS) (see Jul 2013 news and Rogers et al., 2012). Klaus Romero and colleagues at CAMD
Stem Cell Therapies for Neurodegenerative Disease: How Should We Push Ahead?
One was from my colleague Chuck Murry (see Murry et al., 2004), and the other involved Irv Weissman
Adding Dynamics and Nuance to Alzheimer’s Staging
(i.e., their evolution over the course of the disease) (Jack et al., 2010; Weiner et al., 2010; Frisoni
Two New Sets of Diagnostic Criteria—Which Is Right for Your Clinic?
phases (see ARF related news story on McKhann et al., 2011; Albert et al., 2011; and Sperling et al.,
Neurovascular Underpinnings of Alzheimer's Dementia
vascular disease from the diagnosis (McKhann et al., 1984), while vascular dementia was considered
Seeded Aggregation and Transmissible Proteopathy—Creepy Stuff Not Just for Prions Anymore?
can also cause disease characteristics or phenotypes (Collinge et al., 2007). But are prions truly
Not Dead Yet: Estrogen Deserves Another Chance
itself and has brain-specific aromatase (see Sasano et al., 1998). Dominique Toran-Allerand Regarding
Deficiency of Presenilin 1 Inhibits the Normal Cleavage of the Transmembrane Domain of Amyloid Precursor Protein
report by De Strooper et. al., (1),and the accompanying commentary by Haas and Selkoe (2), conclude that
Is Alzheimer’s Disease a Uniquely Human Disorder?
e.g., Bussiere et al., 2003). Brain regions giving rise to long axonal connections have expanded in
Treating Before Symptoms—ADCS Invites Ideas for Clinical Trials in Very Early AD
Petersen criteria for amnestic MCI (Petersen et al., 1999; Petersen, 2004), though the memory impairment
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